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Metabolic & weightLimited humanResearch only

Cagrilintide

AM833

Long-acting amylin analogue that targets satiety through a pathway independent of GLP-1.

Class

Amylin and calcitonin receptor agonist

Half-life

~7–8 days (once-weekly dosing)

Evidence

Limited human data

Limited human data

Small open-label studies, case series, or trials in a narrow population. Directionally informative, not conclusive.

Overview

Amylin is a hormone co-secreted with insulin by pancreatic beta cells. It slows gastric emptying, suppresses glucagon, and signals satiety through the area postrema — overlapping with GLP-1 in outcome but arriving via a distinct receptor system. Cagrilintide is a long-acting analogue built for weekly dosing.

The clinically interesting finding is additivity. Because amylin and GLP-1 act through different receptors, combining them produces more weight loss than either alone. The CagriSema combination (cagrilintide plus semaglutide) reported roughly 22–23% mean weight reduction in phase 2 and phase 3 work, approaching tirzepatide territory.

As a standalone it is less impressive — phase 2 monotherapy produced around 10–11% at 26 weeks. Its real role in the literature is as a combination partner, and it is not approved in any jurisdiction on its own.

How it works

  • Agonises amylin receptors (AMY1–3, calcitonin receptor plus RAMP complexes) in the area postrema.
  • Slows gastric emptying independently of the GLP-1 pathway.
  • Suppresses postprandial glucagon secretion.
  • Reduces homeostatic hunger signalling, complementing GLP-1's effect on food reward.

What the research shows

Researched effects

  • ~10.8% mean weight reduction at 26 weeks as monotherapy (phase 2, 4.5 mg)
  • ~22–23% mean weight reduction combined with semaglutide (CagriSema program)
  • Improved glycemic control in combination regimens
  • Additive satiety effect with GLP-1 agonists due to non-overlapping receptor pathway

Limitations & what it won't do

  • Not approved anywhere as monotherapy or in combination at time of writing.
  • Monotherapy efficacy is well below tirzepatide or semaglutide.
  • Long-term safety data is limited to phase 2/3 trial duration.
  • Research-chemical sourcing carries the usual identity and purity risk.

Dosing protocols

These ranges reflect published trials and community protocols. They are reference information, not a prescription. Doses are per injection unless noted.

Phase 2 monotherapy titration (reference only)

0.3 mg → 4.5 mg
FrequencyOnce weekly
RouteSubcutaneous injection
Cycle26 weeks in the trial protocol

Doubling every 4 weeks: 0.3, 0.6, 1.2, 2.4, 4.5 mg. Documented as trial reference, not a recommendation.

Combination arm (with a GLP-1)

2.4 mg
FrequencyOnce weekly
RouteSubcutaneous injection
CycleContinuous in trial protocols

CagriSema pairs 2.4 mg cagrilintide with 2.4 mg semaglutide. Combination use compounds GI side effects and should only happen under clinical supervision.

Reconstitution & measuring your dose

Handling

Typical vial sizes
5 · 10 mg
Suggested BAC water
2 mL
Storage
Lyophilised powder refrigerated at 2–8 °C, protected from light.
After reconstitution
Reconstituted: approximately 28 days refrigerated.

Why 2 mL? A 10 mg vial in 2 mL yields 5 mg/mL — 0.3 mg lands on 6 units and 4.5 mg on 90 units of a U-100 syringe.

Dose calculator

Draw to

6units

= 0.06 mL · 50 mcg per unit

0u100u

Concentration

5000 mcg/mL

Doses per vial

33

Side effects

Common

  • Nausea
  • Vomiting
  • Constipation
  • Decreased appetite
  • Injection-site reactions
  • Fatigue

Serious / rare

  • Severe GI intolerance when combined with a GLP-1
  • Gallbladder disease with rapid weight loss
  • Hypoglycemia in combination with insulin secretagogues

Contraindications & interactions

Do not use if you have

  • Pregnant, breastfeeding, or trying to conceive

    No pregnancy safety data. Absolute contraindication.

  • Pancreatitis history

    Delayed gastric emptying and rapid weight loss in someone with prior pancreatitis is an unacceptable risk profile.

Use caution if you have

  • Gastroparesis or delayed gastric emptying

    Slowed gastric emptying is a primary mechanism — additive with existing gastroparesis.

  • Gallbladder disease or gallstones

    Rapid weight loss raises gallstone risk.

  • Type 1 diabetes

    Amylin analogues have been used in type 1 diabetes (pramlintide) but require careful insulin coordination.

  • Medullary thyroid carcinoma or MEN2 (personal or family)

    Calcitonin receptor agonism in the context of thyroid C-cell disease is not well characterised — avoid pending clinician review.

Medication interactions

  • avoidInsulinAmylin analogues require insulin dose reduction to avoid severe hypoglycemia — this is prescriber territory.
  • avoidSulfonylureasAdditive hypoglycemia risk.
  • cautionGLP-1 / GIP agonistsCombination is the studied use case and is additive for efficacy, but GI side effects compound substantially. Clinical supervision only.
  • monitorThyroid hormoneDelayed gastric emptying can alter levothyroxine absorption.

What to monitor

  • Weight and body composition
  • Fasting glucose, particularly if on insulin
  • GI tolerability during titration
  • Gallbladder symptoms during rapid loss phases

Commonly combined with

References

Research & educational information only — not medical advice.

You must be 18 or older to use this site. The peptides described are presented as research chemicals intended for research purposes only. Most are not approved by the FDA for human use. Dosing ranges reflect what has appeared in the scientific literature or community protocols and are not prescriptions. Nothing here replaces evaluation by a licensed physician who knows your full medical history.

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